As others have concluded (50, 51) a multimodal attack on all the various growth enhancing systems active in glioblastoma may be required. This paper recommends research leading to a single step toward that goal- aprepitant and maraviroc synergistic inhibition of that portion of ERK1/2 activation that is contributed by NK-1R and CCR5 signaling. The multiple redundant systems evident even in this re

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